Suprascapular Neuropathy and Its Symptoms

neuropathySuprascapular neuropathy is typically the result of traction damage to the Suprascapular nerve. This nerve arises from the upper part of the brachial plexus, the large number of nerves where they exit the spine at the base of the neck, and travels under the trapezius to the scapular where it supplies the Supraspinatus and Infraspinatus muscles.

Suprascapular Neuropathy and Its Symptoms

Damage to this nerve usually occurs during sports that involve overhead movements. Some of these sports include tennis, cricket and volleyball. Injury to the nerve can happen because of compression, traction or direct trauma. Unnatural movements of the scapula can also cause stretching of the nerve. The growth of cysts resulting from superior glenoid labral tears can compress the nerve as well.

If you think you are suffering from suprascapular neuropathy, you’ll be experiencing these symptoms:

  • Aching or burning pain of the shoulder joint.
  • Deep pain within the shoulder joint.
  • Pain that radiates through the arm.
  • Gradually occurring pain.
  • Weakness of the shoulder joint into abduction – lifting the arm out to the side – and external rotation.
  • Wasting of the Supraspinatus and Infraspinatus muscles.
Enhanced by Zemanta

How Tai Chi Helps with Peripheral Neuropathy

If you’ve driven past a park and seen a group of people performing carefully synchronized movements, they were most likely practicing the ancient Chinese martial art Tai Chi. This martial art is supposed to help enhance mental and physical health, so it’s no wonder that it was found to be able to help improve symptoms or peripheral neuropathy.

How Tai Chi Helps with Peripheral Neuropathy

But before you can understand how Tai Chi can help with this condition, you first need to understand what peripheral neuropathy is and how it affects those who are infected with it. Peripheral neuropathy is a systemic degenerative disease that directly affects your peripheral nervous system, which is responsible for carrying the signals from your brain and spinal column to everywhere else in your body. When this happens, you begin losing the ability to move your extremities and feel pain in your hands and feet. Unfortunately, there isn’t a cure for this condition, but the practice of Tai Chi can really help improve the symptoms that come along with it.

Patients who have stayed active through Tai Chi have reported that their episodes of severe burning and pain that they experienced on a daily basis were reduced to maybe one or two episodes a year. That’s a pretty significant amount if you ask me. The benefits of Tai Chi don’t stop there, though. It also helps with strength and balance as well as helps calm the mind and body.

Enhanced by Zemanta

Benfotiamine Product Summary 7 of 7 – The End of an AGE

Benfotiamine Product Summary 7 of 7 – The End of an AGE

These are not test-tube studies. The results experienced when taking Benfotiamine occur not merely in labs, but in lives: in the bodies – and in the health – of living things, from experimental animals to human beings. In Benfotiamine, we finally have a proven way to protect tissues from the AGE assault.

Enhanced by Zemanta

Benfotiamine Product Summary 6 of 7 – Benfotiamine in Other Vulnerable Tissues

Source: www.benfotiamine.org

Benfotiamine Product Summary 6 of 7 – Benfotiamine in Other Vulnerable Tissues

More recently, new studies have begun to document Benfotiamine’s ability to shield other tissues from AGE damage. One just-published study tested the ability of thiamin and Benfotiamine to protect diabetic rodents’ retinas from the ravages of AGE.

The researchers then gave one group of diabetic rodents Benfotiamine supplements, and left another group unsupplemented, keeping a third group of nondiabetic animals as a control group. Nine months later, they examined the animals’ eyes, testing the level of AGE in their retinas, examining metabolic abnormalities of the cells, and looking for acellular capillaries (the dead husks left behind when the cells of the tiny blood vessels of the eye die).

Benfotiamine supplements normalized AGE levels in the diabetics’ retina, as well as several key metabolic parameters within the diabetic animals’ cells – without influencing body weight or blood sugar (as measured by HbA1c). More importantly, Benfotiamine prevented the AGE-associated retinal damage. After nine months of diabetes, diabetic animals had suffered three times as many acellular capillaries as were found in healthy animals. But with the protection afforded by Benfotiamine, the number of acellular capillaries in the supplemented diabetics was indistinguishable from that of their normal, healthy cousins!

And there’s another AGE-related disease that researchers believe Benfotiamine may fight: the loss of kidney function which accompanies “normal” aging, and which is accelerated by diabetes. Dr. Paul Thornalley of the University of Essex has just completed a study designed to see if Benfotiamine will protect diabetic rodents against kidney damage. While the results have not yet been published, Dr. Thornalley has indicated that both megadose thiamin and Benfotiamine caused clear-cut reductions in the leakage of protein – with Benfotiamine showing itself to be the superior intervention. A second study is now underway to see if Benfotiamine will actually improve kidney function in diabetic animals with pre-existing kidney damage, as it has already been shown to do in the nerves of diabetic animals and humans.

Go to Part 7

Enhanced by Zemanta

Benfotiamine Product Summary 5 of 7 – Shielding Nerve Structure

Source: http://www.benfotiamine.org/Benfotiamine.htm

Benfotiamine Product Summary 5 of 7 – Shielding Nerve Structure

While most “anti-AGE” supplements rely on test-tube “browning” experiments as the “evidence” of efficacy, Benfotiamine has been proven in multiple real-world human and animal studies to reduce AGE formation and support tissue structure and function in diabetics.

Most impressively, many randomized, double-blind, placebo-controlled human trials have proven that Benfotiamine powerfully supports nerve function in diabetic neuropathy. In one trial, 24 people suffering with diabetic neuropathy took either Benfotiamine (plus doses of common B6 and B12 similar to those used in multivitamins) or a look-alike dummy pill, spread out into three pills over the course of the day, for twelve weeks. The participants started with 320 milligrams of Benfotiamine per day for the first two weeks, followed by 120 milligrams for the rest of the trial. Before and after the trial, the function of patients’s nerve cells were tested using nerve conduction velocity (NCV) and vibratory perception threshold (which tests the nerves’s sensitivity by determining the lowest level at which vibrations applied at key nerve sites are first felt).

At the end of the trial, the vibration perception threshold had “clearly” improved by 30% in those who had taken the Benfotiamine supplements, while it had worsened in the placebo group by 5% at one site and by 32% at another. At the same time, people taking Benfotiamine experienced statistically significant improvements in nerve conduction velocity from the feet, even as this aspect of nerve function deteriorated in those taking the look-alike pills!

The power of Benfotiamine to improve vibratory perception threshold and nerve conduction velocity have been confirmed in other trials. Clinical trials have also shown that Benfotiamine supports nerve function in diabetics as measured by many other methods. For instance, Benfotiamine users experience a 50% reduction in diabetic nerve pain, along with an increased ability of the nerves to detect an electrical current, respond to electrical stimulation, and regulate the heartbeat. Similarly, Benfotiamine prevents this loss of control from happening in the first place in diabetic dogs. In another human clinical trial, a B-vitamin combination using Benfotiamine as its thiamin source was put head-to-head with a B-complex supplement that included a mega dose of conventional thiamin. Benfotiamine proved its effectiveness on several of these key parameters, while the standard thiamin pill failed.

These benefits are not due to changes in blood sugar levels (either fasting, or after a meal, or averaged over several months (as measured by HbA1c), or improvements in metabolic benchmarks. They are the direct results of Benfotiamine’s AGE-fighting, metabolic-balancing powers.

Go to Part 6

Enhanced by Zemanta

Benfotiamine Product Summary 4 of 7 – Benfotiamine: the TPP Solution

Source: http://www.benfotiamine.org/Benfotiamine.htm

Benfotiamine Product Summary 4 of 7 – Benfotiamine: the TPP Solution

Fortunately, an effective way to boost thiamin pyrophosphate in your cells does exist: Benfotiamine (S-benzoylthiamine-O-monophosphate). Benfotiamine is the most potent of the allithiamines, a unique class of thiamin-derived compounds present in trace quantities in roasted crushed garlic and other vegetables from the Allium genus (such as onions, shallots, and leeks). Benfotiamine’s unique open-ringed structure makes it able to pass directly through cell membranes, readily crossing the intestinal wall and being taken straight into the cell.

As a result, your body absorbs Benfotiamine better than thiamin itself, and levels of thiamin and TPP remain higher for longer. Thiamin absorption from Benfotiamine is about five times as great as from conventional thiamin supplements. And the effect is even more impressive at the tissue level: brain and muscle, for instance, take in five- to twentyfive-fold as much thiamin in the form of allithiamines as they do of an equal amount of regular thiamin. And Benfotiamine is even more bioavailable than the other allithiamines, including thiamin tetrahydrofurfuryl disulfide/TTFD. Yet Benfotiamine is actually less toxic than conventional thiamin supplements!

By effectively increasing levels of thiamin itself, Benfotiamine dramatically boosts AGE-fighting thiamin pyrophosphate and cell-shielding transketolase activity in your body.

Go to Part 5

Enhanced by Zemanta

Benfotiamine Product Summary 3 of 7 – TPP: Our Hero … in Chains!

Source: http://www.benfotiamine.org/Benfotiamine.htm

Benfotiamine Product Summary 3 of 7 – TPP: Our Hero … in Chains!

There is a nutrient that could, in theory, pack a potent wallop against the AGE onslaught: Thiamin Pyrophosphate (TPP), the active coenzyme form of the B-complex vitamin thiamin. In 1996, researchers showed that TPP could step in to stop AGE formation at the most important point in the process: the late, irreversible conversion of Amadori products into full-blown AGEs. What’s more, TPP can exert a two-pronged AGE-inhibiting effect in the body, because boosting TPP in cells stressed by high glucose concentrations opens up an important biochemical “safety valve” in the normal metabolism of blood sugar through an enzyme known as transketolase. Activating transketolase allows the body to shunt excess triosephosphates into a safe alternative metabolic pathway, preventing the logjam that leads to the buildup of triosephosphates and the formation of AGE.

Unfortunately, this does not mean that loading up on regular thiamin (vitamin B1) will free you from glycation’s sticky shackles. The problem is that your body’s ability to absorb and metabolize conventional thiamin supplements is very limited. In fact, no matter how much thiamin you take, you don’t materially increase plasma levels beyond what you get from the first 12 milligrams of the dose. And then getting thiamin into the cells to do its job is just as tricky.

You might think that you can get around this problem by taking supplements containing TPP itself, instead of plain old thiamin. Unfortunately, as part of the normal cellular absorption process, specific enzymes actually strip TPP of its phosphate groups. As a result, you get no additional AGE-battling benefit from taking preformed thiamin pyrophosphate instead of standard thiamin. In fact, when you take supplements based on TPP itself, studies show that thiamin levels and biological activity are actually lower than if you take the same amount of regular thiamin!

Go to Part 4

Enhanced by Zemanta

Benfotiamine Product Summary 2 of 7 – Make that: AGEing you.

Source: www.benfotiamine.org

Amazing product.

One of the most affective products I have tried on the market. . Online Canadian Pharmacy provides one of quickest and easiest ordering processes available oline today.

Benfotiamine Product Summary 2 of 7 – Make that: AGEing you. 

Two Ways to AGE
There are two major ways that AGEs can form inside the body. One way is through a simple series of chemical reactions known as the “Maillard Pathway,” known from food chemistry for a century.

But more recently, scientists have come to understand another pathway of AGE formation – a distinctly biological pathway, which only occurs within your cells because of the body’s metabolism of carbohydrates.

When blood sugar levels rise, some key kinds of cell – including- nerve cells (neurons) and the cells that make up the fine blood cells of the retina of the eye and the filtering units (glomeruli) of the kidney – are also flooded with glucose. The resulting high sugar levels within these cells cause a logjam in the normal cellular metabolism of glucose. This backlog results in a buildup within the cell of super-reactive glucose-metabolic intermediates known as triosephosphates. And once that happens, the excess triosephosphates attack the surrounding proteins, lipids, and DNA, causing AGE damage from within the heart of the cell. It’s these cells that are thus the most vulnerable to the complications of diabetes.

Drugs do exist which can inhibit the formation of AGE, but none are available on the market as yet, and one of the most promising candidate (aminoguanidine) has shown signs of toxicity in human trials and appears to have been abandoned by its developers. On the other hand, some companies are selling supplements marketed as “AGE-inhibitors.” But while many of these herbs and other nutrients may be valuable, and may even inhibit AGEing in a test tube, there’s no evidence that most of these “AGE-blocking” ingredients have any effect on AGEing in your body at the dosages used. Examples include thyme extract, inositol, acetyl-L-carnitine, and a whole host of antioxidants (including NAC and flavonoids, such as quercetin and resveratrol).

Go to Part 3

Enhanced by Zemanta

Benfotiamine Product Summary 1 of 7 – Benfotiamine and the Caramelization of the Flesh

Source: www.benfotiamine.org

Compared to others on the market, best price and quality. ? Generic drugs are copies of brand-name drugs that have exactly the same dosage.

PRODUCT SUMMARY: (Compiled by Advanced Orthomolecular Research (AOR) of Canada)

Benfotiamine and the Caramelization of the Flesh

Benfotiamine and the “Caramelization of the Flesh” – Advanced Glycation Endproducts, or “AGEs” as they’re appropriately called, are the end result of the complex chemical process through which the structure of proteins is warped by exposure to sugars or by other, much more reactive molecules. AGE chemistry is the cause of the “browning” you see when you roast a chicken or make toast, but the same “browning” chemistry is at work in your body every day of your life. In your arteries. In your kidneys. In your heart, your eyes, your skin, your nerves. In every cell, the sugar that your body uses for fuel is busily at work at this very moment, caramelizing your body through exactly the same chemical processes that caramelize onions or peanut brittle.

Glycation math is simple: more sugar equals more AGEd proteins. As a result, people with diabetes begin to feel the effects of glycation at much younger ages than do people with more normal blood sugar levels. Watching people with diabetes age is like watching “normal” aging played on fast-forward. Slowly, imperceptibly, AGE reactions create chemical handcuffs, which gum up your proteins, deactivate your enzymes, trigger unhealthy biochemical signaling in your cells, and damage your DNA. Aging you.

Go to Part 2

Enhanced by Zemanta

Prevention of cardiac autonomic neuropathy in dogs with Benfotiamine.

Source: www.benfotiamine.org

So far so good.

! That is why we pay attention to the quality of the medications.

Prevention of cardiac autonomic neuropathy in dogs with Benfotiamine.


Koltai MZ.
In Gries FA, Federlin K.

Benfotiamin in the Therapy of Polyneuropathy.
New York: Georg Thieme Verlag, 1998; 45-9.

Experimentally-induced diabetes of the dog leads to disturbances in the autonomous neurological function of the heart after approximately 3 months of continuously- observed diabetes. As signs of autonomic cardiac neuropathy, the heart rate variability and Valsalva ratio clearly fell in the untreated diabetic animals. Oral benfotiamine, administered from the sixth day after diabetes-induction, prevented or at least delayed these changes. According to the results, treatment with fat-soluble benfotiamine can play an important role in the therapy and prevention of cardiac autonomic neuropathy, apart from any effect on diabetic metabolic

Enhanced by Zemanta